Evaluation of PEGylated exendin-4 released from poly (lactic-co-glycolic acid) microspheres for antidiabetic therapy

J Pharm Sci. 2015 Jan;104(1):72-80. doi: 10.1002/jps.24238. Epub 2014 Nov 18.

Abstract

Peptide-based therapies have the potential to induce antibody formation if the molecules differ from a native human peptide. Several reports have disclosed the occurrence of antibody generation in a patient treated with exenatide. The immune response can be problematic from a clinical stand point, particularly if the antibodies neutralize the efficacy of the biotherapeutic agent or cause a general immune reaction. To overcome this limit, PEGylated exendin-4 analogs were designed and examined for metabolic stability and biological activity. To develop an extended release delivery system for exendin-4 for the safe and effective delivery of bioactive exendin-4 without peptide acylation and immunogenicity, PEGylated exendin-4 was encapsulated into poly (lactic-co-glycolic acid) (PLGA) microspheres by w/o/w double emulsion solvent evaporation method. Peptide-loaded microspheres were characterized in terms of morphology, particle diameter, and peptide encapsulation efficiency. Then, the release profile of the peptide from PLGA microspheres and the acylated products from PLGA polymer degradation was determined. The results obtained showed that the stability of exendin-4 was greatly improved by PEGylation. Moreover, eliminated acylation during PLGA polymer degradation in vitro and reduced immunogenicity in vivo were observed. The findings demonstrate that PEGylated exendin-4-loaded microspheres may be a safe and biocompatible system for clinical development.

Keywords: PEGylation; PLGA; acylation; exendin-4; immunogenicity; microspheres; peptide delivery; polymeric drug delivery systems.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acylation
  • Animals
  • Antibodies / analysis
  • Delayed-Action Preparations / administration & dosage
  • Delayed-Action Preparations / adverse effects
  • Delayed-Action Preparations / pharmacokinetics
  • Delayed-Action Preparations / therapeutic use
  • Diabetes Mellitus, Type 2 / blood
  • Diabetes Mellitus, Type 2 / drug therapy*
  • Diabetes Mellitus, Type 2 / immunology
  • Drug Compounding
  • Drug Hypersensitivity / blood
  • Drug Hypersensitivity / immunology
  • Drug Hypersensitivity / prevention & control*
  • Exenatide
  • Half-Life
  • Hypoglycemic Agents / administration & dosage*
  • Hypoglycemic Agents / adverse effects
  • Hypoglycemic Agents / pharmacokinetics
  • Hypoglycemic Agents / therapeutic use
  • Incretins / administration & dosage*
  • Incretins / adverse effects
  • Incretins / pharmacokinetics
  • Incretins / therapeutic use
  • Injections, Subcutaneous
  • Lactic Acid / adverse effects
  • Lactic Acid / chemistry*
  • Male
  • Mice, Inbred C57BL
  • Mice, Mutant Strains
  • Microspheres
  • Peptides / administration & dosage*
  • Peptides / adverse effects
  • Peptides / pharmacokinetics
  • Peptides / therapeutic use
  • Polyethylene Glycols / adverse effects
  • Polyethylene Glycols / chemistry*
  • Polyglycolic Acid / adverse effects
  • Polyglycolic Acid / chemistry*
  • Polylactic Acid-Polyglycolic Acid Copolymer
  • Random Allocation
  • Rats, Sprague-Dawley
  • Solubility
  • Suspensions
  • Venoms / administration & dosage*
  • Venoms / adverse effects
  • Venoms / pharmacokinetics
  • Venoms / therapeutic use

Substances

  • Antibodies
  • Delayed-Action Preparations
  • Hypoglycemic Agents
  • Incretins
  • Peptides
  • Suspensions
  • Venoms
  • Polylactic Acid-Polyglycolic Acid Copolymer
  • Polyglycolic Acid
  • Lactic Acid
  • Polyethylene Glycols
  • Exenatide