Novel respiratory syncytial virus (RSV) genotype ON1 predominates in Germany during winter season 2012-13

PLoS One. 2014 Oct 7;9(10):e109191. doi: 10.1371/journal.pone.0109191. eCollection 2014.

Abstract

Respiratory syncytial virus (RSV) is the leading cause of hospitalization especially in young children with respiratory tract infections (RTI). Patterns of circulating RSV genotypes can provide a better understanding of the molecular epidemiology of RSV infection. We retrospectively analyzed the genetic diversity of RSV infection in hospitalized children with acute RTI admitted to University Hospital Heidelberg/Germany between October 2012 and April 2013. Nasopharyngeal aspirates (NPA) were routinely obtained in 240 children younger than 2 years of age who presented with clinical symptoms of upper or lower RTI. We analyzed NPAs via PCR and sequence analysis of the second variable region of the RSV G gene coding for the attachment glycoprotein. We obtained medical records reviewing routine clinical data. RSV was detected in 134/240 children. In RSV-positive patients the most common diagnosis was bronchitis/bronchiolitis (75.4%). The mean duration of hospitalization was longer in RSV-positive compared to RSV-negative patients (3.5 vs. 5.1 days; p<0.01). RSV-A was detected in 82.1%, RSV-B in 17.9% of all samples. Phylogenetic analysis of 112 isolates revealed that the majority of RSV-A strains (65%) belonged to the novel ON1 genotype containing a 72-nucleotide duplication. However, genotype ON1 was not associated with a more severe course of illness when taking basic clinical/laboratory parameters into account. Molecular characterization of RSV confirms the co-circulation of multiple genotypes of subtype RSV-A and RSV-B. The duplication in the G gene of genotype ON1 might have an effect on the rapid spread of this emerging RSV strain.

MeSH terms

  • Amino Acid Sequence
  • Child, Preschool
  • Female
  • Genotype*
  • Germany / epidemiology
  • Humans
  • Infant
  • Infant, Newborn
  • Male
  • Molecular Sequence Data
  • Phylogeny
  • Respiratory Syncytial Virus Infections / epidemiology*
  • Respiratory Syncytial Virus Infections / virology*
  • Respiratory Syncytial Virus, Human / classification
  • Respiratory Syncytial Virus, Human / genetics*
  • Seasons*
  • Sequence Alignment
  • Viral Envelope Proteins / chemistry
  • Viral Envelope Proteins / genetics

Substances

  • Viral Envelope Proteins

Associated data

  • GENBANK/KJ710364
  • GENBANK/KJ710365
  • GENBANK/KJ710366
  • GENBANK/KJ710367
  • GENBANK/KJ710368
  • GENBANK/KJ710369
  • GENBANK/KJ710370
  • GENBANK/KJ710371
  • GENBANK/KJ710372
  • GENBANK/KJ710373
  • GENBANK/KJ710374
  • GENBANK/KJ710375
  • GENBANK/KJ710376
  • GENBANK/KJ710377
  • GENBANK/KJ710378
  • GENBANK/KJ710379
  • GENBANK/KJ710380
  • GENBANK/KJ710381
  • GENBANK/KJ710382
  • GENBANK/KJ710383
  • GENBANK/KJ710384
  • GENBANK/KJ710385
  • GENBANK/KJ710386
  • GENBANK/KJ710387
  • GENBANK/KJ710388
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  • GENBANK/KJ710390
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  • GENBANK/KJ710394
  • GENBANK/KJ710395
  • GENBANK/KJ710396
  • GENBANK/KJ710397
  • GENBANK/KJ710398
  • GENBANK/KJ710399
  • GENBANK/KJ710400
  • GENBANK/KJ710401
  • GENBANK/KJ710402
  • GENBANK/KJ710403
  • GENBANK/KJ710404
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  • GENBANK/KJ710407
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  • GENBANK/KJ710415
  • GENBANK/KJ710416
  • GENBANK/KJ710417
  • GENBANK/KJ710418
  • GENBANK/KJ710419
  • GENBANK/KJ710420

Grants and funding

The authors have no support or funding to report.