Growth hormone receptor signaling is dispensable for HSC function and aging

Blood. 2014 Nov 13;124(20):3076-80. doi: 10.1182/blood-2014-05-575308. Epub 2014 Oct 1.

Abstract

Growth hormone receptor (Ghr) signaling is important in a wide variety of cellular processes including aging; however, the role of Ghr signaling in hematopoietic stem cell (HSC) biology remains unexplored. Within the hematopoietic system, Ghr is expressed in a highly HSC-specific manner and is significantly upregulated during aging. Exposure of young and old HSCs to recombinant growth hormone ex vivo led to diminished short-term reconstitution and restored B-cell output from old HSCs. Hematopoietic-specific genetic deletion of Ghr neither impacted steady-state hematopoiesis nor serial transplantation potential. Repeat challenge with 5-fluorouracil showed that Ghr was dispensable for HSC activation and homeostatic recovery in vivo and, after challenge, Ghr-deficient HSCs functioned normally through serial transplantation. Although exogenous Gh induces age-dependent HSC effects, these results indicate that Ghr signaling appears largely dispensable for HSC function and aging.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aging*
  • Animals
  • Cellular Senescence
  • Gene Deletion
  • Gene Expression
  • Growth Hormone / administration & dosage
  • Growth Hormone / metabolism
  • Hematopoiesis*
  • Hematopoietic Stem Cells / cytology*
  • Hematopoietic Stem Cells / metabolism
  • Mice
  • Receptors, Somatotropin / genetics
  • Receptors, Somatotropin / metabolism*
  • Signal Transduction*

Substances

  • Receptors, Somatotropin
  • Growth Hormone