Burn intensive care

Shawn P Fagan; Mary-Liz Bilodeau; Jeremy Goverman

doi:10.1016/j.suc.2014.05.004

Burn intensive care

Surg Clin North Am. 2014 Aug;94(4):765-79. doi: 10.1016/j.suc.2014.05.004. Epub 2014 Jun 16.

Authors

Shawn P Fagan¹, Mary-Liz Bilodeau¹, Jeremy Goverman²

Affiliations

¹ Sumner Redstone Burn Center, Massachusetts General Hospital, 55 Fruit Street, Boston, MA 02114, USA.
² Sumner Redstone Burn Center, Massachusetts General Hospital, 55 Fruit Street, Boston, MA 02114, USA. Electronic address: jgoverman@partners.org.

PMID: 25085087
DOI: 10.1016/j.suc.2014.05.004

Abstract

As a result of continuous development in the treatment of burns, the LD50 (the burn size lethal to 50% of the population) for thermal injuries has risen from 42% total body surface area (TBSA) during the 1940s and 1950s to more than 90% TBSA for young thermally injured patients. This vast improvement in survival is due to simultaneous developments in critical care, advancements in resuscitation, control of infection through early excision, and pharmacologic support of the hypermetabolic response to burns. This article reviews these recent advances and how they influence modern intensive care of burns.

Keywords: Burns; Colloid; Intensive care; Postburn hypermetabolism.

Publication types

Review

MeSH terms

Acute Kidney Injury / etiology
Acute Kidney Injury / therapy
Burns / therapy*
Critical Care / methods*
Fluid Therapy / methods
Humans
Hyperglycemia / etiology
Hyperglycemia / prevention & control
Metabolic Diseases / etiology
Metabolic Diseases / therapy
Respiration, Artificial / methods
Wound Infection / prevention & control