Glycation of fetal hemoglobin reflects hyperglycemia exposure in utero

Diabetes Care. 2014 Oct;37(10):2830-3. doi: 10.2337/dc14-0549. Epub 2014 Jul 10.

Abstract

Objective: The lifetime risk of metabolic diseases in offspring of women with gestational diabetes mellitus (GDM) depends, at least in part, on the impact of glycemic fetal programming. To quantify this impact, we have developed and validated a unique mass spectrometry method to measure the percentage of glycated hemoglobin in cord blood.

Research design and methods: This case-control study includes 37 GDM women and 30 pregnant women with normal glucose tolerance (NGT).

Results: Glycation of the α-chain (Glα) was higher in neonates from GDM (2.32 vs. 2.20%, P < 0.01). Glα strongly correlated with maternal A1C measured at delivery in the overall cohort (r = 0.67, P < 0.0001) as well as in each group (GDM: r = 0.66, P < 0.0001; NGT: r = 0.50, P = 0.01).

Conclusions: Thus, Glα may reflect hyperglycemic exposure during the last weeks of fetal development. Future studies will confirm Glα is a predictive biomarker of prenatally programmed lifetime metabolic health and disease.

Trial registration: ClinicalTrials.gov NCT01025973.

Publication types

  • Clinical Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Blood Glucose / metabolism
  • Case-Control Studies
  • Diabetes, Gestational / blood*
  • Female
  • Fetal Blood / metabolism
  • Fetal Development
  • Fetal Hemoglobin / metabolism*
  • Glycated Hemoglobin / metabolism*
  • Glycosylation
  • Humans
  • Hyperglycemia / blood*
  • Infant, Newborn
  • Male
  • Metabolic Diseases / epidemiology*
  • Metabolic Diseases / etiology
  • Pregnancy
  • Prenatal Exposure Delayed Effects / blood*
  • Prenatal Exposure Delayed Effects / diagnosis
  • Young Adult

Substances

  • Blood Glucose
  • Glycated Hemoglobin A
  • Fetal Hemoglobin

Associated data

  • ClinicalTrials.gov/NCT01025973