Abstract
Macrophage infiltration has been identified as an independent poor prognostic factor in several cancer types. The major survival factor for these macrophages is macrophage colony-stimulating factor 1 (CSF-1). We generated a monoclonal antibody (RG7155) that inhibits CSF-1 receptor (CSF-1R) activation. In vitro RG7155 treatment results in cell death of CSF-1-differentiated macrophages. In animal models, CSF-1R inhibition strongly reduces F4/80(+) tumor-associated macrophages accompanied by an increase of the CD8(+)/CD4(+) T cell ratio. Administration of RG7155 to patients led to striking reductions of CSF-1R(+)CD163(+) macrophages in tumor tissues, which translated into clinical objective responses in diffuse-type giant cell tumor (Dt-GCT) patients.
Trial registration:
ClinicalTrials.gov NCT01494688.
Copyright © 2014 Elsevier Inc. All rights reserved.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Animals
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Antibodies, Monoclonal / immunology*
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Antibodies, Monoclonal / pharmacokinetics
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Antibodies, Monoclonal / pharmacology*
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Antibodies, Monoclonal, Humanized
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Cell Differentiation / physiology
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Cell Line, Tumor
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Clinical Trials, Phase I as Topic
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Cohort Studies
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Colonic Neoplasms / immunology
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Colonic Neoplasms / metabolism
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Colonic Neoplasms / therapy*
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Female
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Humans
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Macaca fascicularis
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Macrophages / cytology
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Macrophages / drug effects*
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Macrophages / immunology*
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Macrophages / metabolism
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Male
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Mice, Inbred C57BL
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Models, Molecular
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Receptor, Macrophage Colony-Stimulating Factor / antagonists & inhibitors*
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Receptor, Macrophage Colony-Stimulating Factor / immunology*
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Receptor, Macrophage Colony-Stimulating Factor / metabolism
Substances
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Antibodies, Monoclonal
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Antibodies, Monoclonal, Humanized
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emactuzumab
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Receptor, Macrophage Colony-Stimulating Factor
Associated data
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ClinicalTrials.gov/NCT01494688