Post-paracentesis circulatory derangements are related to monocyte activation

Liver Int. 2014 Aug;34(7):1001-7. doi: 10.1111/liv.12450. Epub 2014 Mar 3.

Abstract

Background & aims: Post-paracentesis circulatory dysfunction is associated with development of hepatorenal syndrome and increased mortality. The impact of large volume paracentesis (LVP) on the 24-h blood pressure (BP) profile is unknown, and the relationship to Na+-retentive and pro-inflammatory cytokines also remains unknown. The aims of this study were to (i) define the effects of LVP with albumin administration on 24-h BP profiles, and (ii) relate changes in BP over time to changes in Na+-retentive hormones, clinical factors and inflammatory cytokines.

Methods: Ten patients undergoing LVP had 24-h ambulatory BP monitoring performed pre- and post-paracentesis. Markers of the innate immune system, bacterial translocation and Na+-retentive hormones were drawn pre- and post-LVP.

Results: Mean arterial pressure (MAP) dropped in nine of the 10 patients in the 24 h following a paracentesis compared to 24 h preceding the procedure (mean drop of 5.5 mmHg, P<0.005). A mixed effects model was used to define time-covariate interactions in predicting changes in BP profile. Monocyte chemotactic protein-1 (MCP1) was associated with Δsystolic BP (β=-0.011, P<0.05), Δdiastolic BP (β=-0.012, P<0.05) and ΔMAP (β=-0.012, P<0.05). Plasma renin activity was also significantly associated with Δsystolic BP (β=-0.21, P<0.05). Renal function was also significantly reduced following LVP.

Conclusions: Systolic, diastolic and MAP decreased over 24 h after LVP compared to the 24 h pre-LVP. This drop is related to increases in MCP-1 after LVP. Increased MCP-1, a marker of monocyte activation, was strongly related to changes in BP.

Keywords: 24-h ambulatory blood pressure monitoring; cirrhosis; inflammation; paracentesis.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Ascites / etiology
  • Ascites / surgery*
  • Blood Pressure / physiology
  • Cytokines / blood
  • Female
  • Hemodynamics
  • Humans
  • Kidney / metabolism
  • Kidney / physiology
  • Liver Cirrhosis / complications*
  • Liver Cirrhosis / pathology
  • Male
  • Middle Aged
  • Monocytes / physiology*
  • Paracentesis / adverse effects*
  • Prospective Studies
  • Renin / blood
  • Serum Albumin / pharmacology
  • Shock / etiology*
  • Shock / physiopathology*
  • Shock / prevention & control
  • Vasodilation / physiology
  • Virginia

Substances

  • Cytokines
  • Serum Albumin
  • Renin