Abstract
MicroRNA miR-302 has been found to induce some tumor cell lines to "transdifferentiate" into miRNA-induced pluripotent stem cells (mirPS), thereby inhibiting tumor cell proliferation and reducing tumorigenicity. This study firstly found that miR-302 inhibited the proliferation and migration of endometrial cell line, Ishikawa and HEC-1-B, and arrested cell cycle at the G2/M phase. In addition, miR-302 inhibited tumorigenicity in immunodeficient mice transplanted with Ishikawa cells. Microarray and Western blotting results showed that miR-302 significantly inhibited CDK1 and Cyclin D1 gene expression in Ishikawa cells. MiR-302 directly targeted Cyclin D1, but indirectly regulated CDK1 gene expression.
Keywords:
G2/M phase; HEC-1-B cells; Ishikawa cells; MicroRNA miR-302.
Copyright © 2013 Elsevier Ireland Ltd. All rights reserved.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Animals
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Cell Growth Processes / genetics
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Cell Line, Tumor
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Cell Movement / genetics
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Cyclin D1 / antagonists & inhibitors*
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Cyclin D1 / genetics
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Cyclin D1 / metabolism
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Cyclin-Dependent Kinase Inhibitor p57 / antagonists & inhibitors*
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Cyclin-Dependent Kinase Inhibitor p57 / genetics
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Cyclin-Dependent Kinase Inhibitor p57 / metabolism
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Endometrial Neoplasms / genetics
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Endometrial Neoplasms / metabolism
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Endometrial Neoplasms / pathology
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Endometrial Neoplasms / therapy*
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Female
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Gene Expression Profiling
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Genetic Therapy / methods*
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Humans
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Mice
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Mice, Nude
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MicroRNAs / administration & dosage*
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MicroRNAs / genetics
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Signal Transduction
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Transfection / methods
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Xenograft Model Antitumor Assays
Substances
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CDKN1C protein, human
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Cyclin-Dependent Kinase Inhibitor p57
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MIRN302A microRNA, human
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MicroRNAs
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Cyclin D1