Altered phenotype and function of dendritic cells in individuals with chronic periodontitis

Arch Oral Biol. 2013 Sep;58(9):1208-16. doi: 10.1016/j.archoralbio.2013.03.013. Epub 2013 Apr 25.

Abstract

Objective: To investigate the effects of periodontal bacterial lysates on maturation and function of mature monocyte-derived dendritic cells (m-MDDCs) derived from individuals with chronic periodontitis (CP) or healthy periodontal tissue (HP).

Design: m-MDDCs derived from peripheral blood monocytes, cultured for 7 days in presence of interleukin (IL)-4 and granulocyte-macrophage colony stimulating factor (GM-CSF), were stimulated with lysates of Streptococcus sanguinis, Prevotella intermedia, Porphyromonas gingivalis, or Treponema denticola on day 4, and were then phenotyped. IL-10, IL-12 and IFN-gamma concentration in the supernatant of cultures were measured.

Results: Expression of HLA-DR was lower in bacterial-unstimulated mature m-MDDC from CP compared to HP (p=0.04), while expression of CD1a and CD123 were higher in CP. The expression pattern of HLA-DR, CD11c, CD123, and CD1a did not change on bacterial stimulation, regardless of the bacteria. Stimulation with P. intermedia upregulated CD80 and CD86 in CP cells (p≤0.05). Production of IL-12p70 by bacterial-unstimulated m-MDDCs was 5.8-fold greater in CP compared to HP. Bacterial stimulation further increased IL-12p70 production while decreasing IL-10. Significantly more IFN-gamma was produced in co-cultures of CP m-MDDCs than with HP m-MDDCs when cells were stimulated with P. intermedia (p=0.009).

Conclusions: Bacterial-unstimulated m-MDDC from CP exhibited a more immature phenotype but a cytokine profile biased towards proinflammatory response; this pattern was maintained/exacerbated after bacterial stimulation. P. intermedia upregulated co-stimulatory molecules and IFN-gamma expression in CP m-MDDC. These events might contribute to periodontitis pathogenesis.

Keywords: Immune response; Periodontal bacteria; Periodontal disease.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antigens, CD / metabolism*
  • Cell Extracts
  • Chronic Periodontitis / blood
  • Chronic Periodontitis / genetics*
  • Chronic Periodontitis / immunology
  • Coculture Techniques
  • Cytokines / biosynthesis*
  • Dendritic Cells / immunology
  • Dendritic Cells / metabolism*
  • Flow Cytometry
  • Granulocyte-Macrophage Colony-Stimulating Factor / immunology*
  • HLA-DR Antigens / metabolism*
  • Humans
  • Interleukin-4 / immunology*
  • Periodontium / microbiology
  • Periodontium / pathology
  • Phenotype
  • Porphyromonas gingivalis / pathogenicity
  • Prevotella intermedia / pathogenicity
  • Streptococcus sanguis / pathogenicity
  • Treponema denticola / pathogenicity

Substances

  • Antigens, CD
  • Cell Extracts
  • Cytokines
  • HLA-DR Antigens
  • Interleukin-4
  • Granulocyte-Macrophage Colony-Stimulating Factor