Enrichment of colorectal cancer stem cells through epithelial-mesenchymal transition via CDH1 knockdown

Mol Med Rep. 2012 Sep;6(3):507-12. doi: 10.3892/mmr.2012.938. Epub 2012 Jun 7.

Abstract

Cancer stem cells (CSCs) are considered to be the origin of cancer relapse and metastasis. The better understanding of CSCs, including CSCs in human colorectal cancer (CRC), may facilitate prevention and treatment. This study aimed to establish a CSC enrichment model via the induction of epithelial-mesenchymal transition (EMT) in CRC cells. We established an EMT model using the SW480 CRC cell line by CDH1 knockdown using shRNA interference. CD24+CD44+ cells were enriched in the CDH1 knockdown cells. The cells exhibited mesenchymal morphology and expressed high levels of EMT-related proteins, which confirmed that these cells had undergone EMT. Our results further showed that the proliferation rate of the transfected cells was reduced, whereas their colony-forming capacity and tumorigenesis in vivo was significantly enhanced compared to the control cells. In conclusion, these cells were highly enriched CSCs (compared to normal CSCs) and may be used as a stable model for cancer research and anticancer drug screening.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antigens, CD
  • CD24 Antigen / metabolism
  • Cadherins / antagonists & inhibitors
  • Cadherins / genetics
  • Cadherins / metabolism*
  • Cell Line, Tumor
  • Cell Transformation, Neoplastic
  • Colorectal Neoplasms / metabolism
  • Colorectal Neoplasms / pathology*
  • Epithelial-Mesenchymal Transition
  • Humans
  • Hyaluronan Receptors / metabolism
  • Mice
  • Mice, Nude
  • Neoplastic Stem Cells / cytology
  • Neoplastic Stem Cells / metabolism*
  • RNA Interference
  • RNA, Small Interfering / metabolism
  • Transplantation, Heterologous

Substances

  • Antigens, CD
  • CD24 Antigen
  • CDH1 protein, human
  • Cadherins
  • Hyaluronan Receptors
  • RNA, Small Interfering