Tumor-targeted T cells modified to secrete IL-12 eradicate systemic tumors without need for prior conditioning

Blood. 2012 May 3;119(18):4133-41. doi: 10.1182/blood-2011-12-400044. Epub 2012 Feb 21.

Abstract

Adoptive cell therapy with tumor-targeted T cells is a promising approach to cancer therapy. Enhanced clinical outcome using this approach requires conditioning regimens with total body irradiation, lymphodepleting chemotherapy, and/or additional cytokine support. However, the need for prior conditioning precludes optimal application of this approach to a significant number of cancer patients intolerant to these regimens. Herein, we present preclinical studies demonstrating that treatment with CD19-specific, chimeric antigen receptor (CAR)-modified T cells that are further modified to constitutively secrete IL-12 are able to safely eradicate established disease in the absence of prior conditioning. We demonstrate in a novel syngeneic tumor model that tumor elimination requires both CD4(+) and CD8(+) T-cell subsets, autocrine IL-12 stimulation, and subsequent IFNγ secretion by the CAR(+) T cells. Importantly, IL-12-secreting, tumor-targeted T cells acquire intrinsic resistance to T regulatory cell-mediated inhibition. Based on these preclinical data, we anticipate that adoptive therapy using CAR-targeted T cells modified to secrete IL-12 will obviate or reduce the need for potentially hazardous conditioning regimens to achieve optimal antitumor responses in cancer patients.

Publication types

  • Evaluation Study
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antigens, CD19 / genetics
  • Antigens, CD19 / immunology
  • Antineoplastic Agents, Alkylating / therapeutic use
  • B-Lymphocytes / drug effects
  • B7-1 Antigen / genetics
  • Combined Modality Therapy
  • Cyclophosphamide / therapeutic use
  • Cytotoxicity, Immunologic
  • Humans
  • Immunotherapy, Adoptive*
  • Interferon-gamma / blood
  • Interleukin-12 / blood
  • Interleukin-12 / genetics
  • Interleukin-12 / metabolism*
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Mice, Transgenic
  • Recombinant Fusion Proteins / genetics
  • T-Cell Antigen Receptor Specificity
  • T-Lymphocyte Subsets / immunology
  • T-Lymphocyte Subsets / metabolism
  • T-Lymphocyte Subsets / transplantation*
  • T-Lymphocytes, Regulatory / drug effects
  • T-Lymphocytes, Regulatory / immunology
  • Thymoma / drug therapy
  • Thymoma / immunology
  • Thymoma / therapy*
  • Thymus Neoplasms / drug therapy
  • Thymus Neoplasms / immunology
  • Thymus Neoplasms / therapy*
  • Transplantation Conditioning*
  • Transplantation, Isogeneic

Substances

  • Antigens, CD19
  • Antineoplastic Agents, Alkylating
  • B7-1 Antigen
  • Recombinant Fusion Proteins
  • Interleukin-12
  • Interferon-gamma
  • Cyclophosphamide