Inhibition of thrombin by synthetic hirudin peptides

FEBS Lett. 1990 Sep 17;270(1-2):85-9. doi: 10.1016/0014-5793(90)81240-o.

Abstract

To investigate the role of different regions of hirudin in the interaction with the proteinase thrombin, segments of hirudin containing 15-51 residues were synthesized. The C-terminal segment 40-65 inhibited the fibrinogen clotting activity of thrombin but not amidolysis of tosyl-Gly-Pro-Arg-p-nitroanilide. Central peptide 15-42 was insoluble at pH 7, but peptide 15-65 inhibited fibrinogen clotting and amidolysis to an equal extent. The N-terminal loop peptide 1-15 had no inhibitory activity and did not affect the potency of peptide 15-65. These data suggest that the central region inhibits catalysis.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Amino Acid Sequence
  • Hirudins / analogs & derivatives*
  • Hirudins / chemical synthesis
  • Hirudins / pharmacology
  • Molecular Sequence Data
  • Peptide Fragments / chemical synthesis
  • Peptide Fragments / pharmacology
  • Structure-Activity Relationship
  • Thrombin / antagonists & inhibitors*

Substances

  • Hirudins
  • Peptide Fragments
  • Thrombin