Vascular effects of glycoprotein130 ligands--part I: pathophysiological role

Vascul Pharmacol. 2012 Jan-Feb;56(1-2):34-46. doi: 10.1016/j.vph.2011.12.004. Epub 2011 Dec 16.

Abstract

The vessel wall is no longer considered as only an anatomical barrier for blood cells but is recognized as an active endocrine organ. Dysfunction of the vessel wall occurs in various disease processes including atherosclerosis, hypertension, peripheral artery disease, aneurysms, and transplant and diabetic vasculopathies. Different cytokines were shown to modulate the behavior of the cells, which constitute the vessel wall such as immune cells, endothelial cells and smooth muscle cells. Glycoprotein 130 (gp130) is a common cytokine receptor that controls the activity of a group of cytokines, namely, interleukin (IL)-6, oncostatin M (OSM), IL-11, ciliary neurotrophic factor (CNTF), leukemia inhibitory factor (LIF), cardiotrophin-1 (CT-1), cardiotrophin-like cytokine (CLC), IL-27, and neuropoietin (NP). Gp130 and associated cytokines have abundantly diverse functions. Part I of this review focuses on the pathophysiological functions of gp130 ligands. We specifically describe vascular effects of these molecules and discuss the respective underlying molecular and cellular mechanisms.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Blood Vessels / metabolism*
  • Blood Vessels / pathology*
  • Cytokine Receptor gp130 / metabolism*
  • Cytokines / metabolism*
  • Humans
  • Ligands
  • Vascular Diseases / metabolism*
  • Vascular Diseases / pathology*

Substances

  • Cytokines
  • Ligands
  • Cytokine Receptor gp130