[DNA damage caused by suicide gene therapy system under Tet-On regulation in breast cancer cells]

Zhong Nan Da Xue Xue Bao Yi Xue Ban. 2011 Sep;36(9):836-43. doi: 10.3969/j.issn.1672-7347.2011.09.004.
[Article in Chinese]

Abstract

Objective: To determine the effect and molecular mechanism of DNA damage caused by suicide gene therapy system HSV-TK/GCV under Tet-On regulation in human breast cancer cell line MCF-7 infected by recombinant adeno-associated virus (rAAV).

Methods: We used comet assay to detect the effect of HSV-TK/GCV suicide gene regulation system on MCF-7 DNA damage, and analyzed the expression change of relative DNA damage response active genes and proteins with RT-PCR and Western blot.

Results: Compared with other control groups, the comet assay showed that MCF-7 cells with HSV-TK/GCV treatment had obvious comet tails, and the expression level of DNA damage response active genes and proteins changed obviously in the HSV-TK/GCV treatment group,such as ATM, p53 and p27,but CyclinE and CDK2 did not change.

Conclusion: DNA damage on MCF-7 cells is resulted from HSV-TK/GCV in suicide gene therapy system through a p53-dependent signal pathway, causing cell cycle arrest and cell death.

Publication types

  • English Abstract
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Breast Neoplasms / genetics*
  • Breast Neoplasms / pathology
  • Breast Neoplasms / therapy
  • DNA Damage*
  • Dependovirus / genetics
  • Female
  • Ganciclovir / metabolism
  • Ganciclovir / pharmacology
  • Gene Expression Regulation, Neoplastic
  • Genes, Transgenic, Suicide / genetics*
  • Genetic Therapy*
  • Humans
  • MCF-7 Cells
  • Recombinant Fusion Proteins / genetics
  • Recombinant Fusion Proteins / metabolism
  • Simplexvirus / enzymology
  • Thymidine Kinase / genetics

Substances

  • Recombinant Fusion Proteins
  • Thymidine Kinase
  • Ganciclovir