Crosstalk between NOTCH and AKT signaling during murine megakaryocyte lineage specification

Blood. 2011 Aug 4;118(5):1264-73. doi: 10.1182/blood-2011-01-328567. Epub 2011 Jun 7.

Abstract

The NOTCH signaling pathway is implicated in a broad range of developmental processes, including cell fate decisions. However, the molecular basis for its role at the different steps of stem cell lineage commitment is unclear. We recently identified the NOTCH signaling pathway as a positive regulator of megakaryocyte lineage specification during hematopoiesis, but the developmental pathways that allow hematopoietic stem cell differentiation into the erythro-megakaryocytic lineages remain controversial. Here, we investigated the role of downstream mediators of NOTCH during megakaryopoiesis and report crosstalk between the NOTCH and PI3K/AKT pathways. We demonstrate the inhibitory role of phosphatase with tensin homolog and Forkhead Box class O factors on megakaryopoiesis in vivo. Finally, our data annotate developmental mechanisms in the hematopoietic system that enable a decision to be made either at the hematopoietic stem cell or the committed progenitor level to commit to the megakaryocyte lineage, supporting the existence of 2 distinct developmental pathways.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Differentiation* / genetics
  • Cell Differentiation* / physiology
  • Cell Lineage / genetics
  • Cell Lineage / physiology*
  • Cells, Cultured
  • Forkhead Transcription Factors / genetics
  • Forkhead Transcription Factors / metabolism
  • Forkhead Transcription Factors / physiology
  • Megakaryocytes / metabolism
  • Megakaryocytes / physiology*
  • Mice
  • Mice, Inbred C57BL
  • Mice, Transgenic
  • Oncogene Protein v-akt / genetics
  • Oncogene Protein v-akt / metabolism*
  • Oncogene Protein v-akt / physiology
  • PTEN Phosphohydrolase / genetics
  • PTEN Phosphohydrolase / metabolism
  • PTEN Phosphohydrolase / physiology
  • Receptor Cross-Talk / physiology
  • Receptors, Notch / genetics
  • Receptors, Notch / metabolism*
  • Receptors, Notch / physiology
  • Signal Transduction / genetics
  • Signal Transduction / physiology
  • Thrombopoiesis / genetics

Substances

  • Forkhead Transcription Factors
  • Receptors, Notch
  • Oncogene Protein v-akt
  • PTEN Phosphohydrolase