Abstract
The incorporation of R,R-1,2-diaminocyclohexane at C4 in a series of 2,4-diaminopyrimidines led to a number of ALK inhibitors in which optimized activity was achieved by conversion of the 2-amino group into a methanesulfonamide. Tumor growth inhibition was observed when an orally bioavailable analog was evaluated in a Karpas-299 tumor xenograft mouse model.
Copyright © 2011 Elsevier Ltd. All rights reserved.
MeSH terms
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Administration, Oral
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Anaplastic Lymphoma Kinase
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Animals
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Antineoplastic Agents / administration & dosage
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Antineoplastic Agents / chemical synthesis*
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Antineoplastic Agents / pharmacology
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Cyclohexylamines / administration & dosage
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Cyclohexylamines / chemical synthesis*
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Cyclohexylamines / pharmacology
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Enzyme Activation / drug effects
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Enzyme Inhibitors / administration & dosage
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Enzyme Inhibitors / chemical synthesis
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Enzyme Inhibitors / pharmacology
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Infusion Pumps
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Inhibitory Concentration 50
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Mice
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Molecular Structure
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Neoplasms / drug therapy
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Pyrimidines / administration & dosage
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Pyrimidines / chemical synthesis*
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Pyrimidines / pharmacology
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Rats
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Rats, Sprague-Dawley
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Receptor Protein-Tyrosine Kinases / antagonists & inhibitors*
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Xenograft Model Antitumor Assays
Substances
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Antineoplastic Agents
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Cyclohexylamines
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Enzyme Inhibitors
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Pyrimidines
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2,4-diaminopyrimidine
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Alk protein, mouse
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Alk protein, rat
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Anaplastic Lymphoma Kinase
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Receptor Protein-Tyrosine Kinases