Global cerebral edema and brain metabolism after subarachnoid hemorrhage

Stroke. 2011 Jun;42(6):1534-9. doi: 10.1161/STROKEAHA.110.604488. Epub 2011 Apr 14.

Abstract

Background and purpose: Global cerebral edema is common among patients with poor-grade subarachnoid hemorrhage and is associated with poor outcome. Currently no targeted therapy exists largely due to an incomplete understanding of the underlying mechanisms.

Methods: This is a prospective observational study including 39 consecutive patients with poor-grade subarachnoid hemorrhage with multimodal neuromonitoring. Levels of microdialysate lactate-pyruvate ratio, episodes of cerebral metabolic crisis (lactate-pyruvate ratio >40 and brain glucose <0.7 mmol/L), brain tissue oxygen tension, cerebral perfusion pressure, and transcranial Doppler sonography flow velocities were analyzed.

Results: Median age was 54 years (range, 45 to 61 years) and 62% were female. Patients with global cerebral edema on admission (n=24 [62%]) had a higher incidence of metabolic crisis in the first 12 hours of monitoring (n=15 [15% versus 2%], P<0.05) and during the total time of neuromonitoring (20% versus 3%, P<0.001) when compared to those without global cerebral edema. There was no difference in brain tissue oxygen tension or cerebral perfusion pressure between the groups; however, in patients with global cerebral edema, a higher cerebral perfusion pressure was associated with lower lactate-pyruvate ratio (P<0.05). Episodes of metabolic crisis were associated with poor outcome (modified Rankin Scale score 5 or 6, P<0.05).

Conclusions: In patients with poor-grade subarachnoid hemorrhage, global cerebral edema is associated with early brain metabolic distress.

MeSH terms

  • Brain / metabolism*
  • Brain Edema / etiology*
  • Brain Edema / physiopathology
  • Female
  • Glucose / metabolism
  • Humans
  • Lactic Acid / metabolism
  • Middle Aged
  • Oxygen / metabolism
  • Prospective Studies
  • Pyruvic Acid / metabolism
  • Subarachnoid Hemorrhage / complications*
  • Subarachnoid Hemorrhage / physiopathology

Substances

  • Lactic Acid
  • Pyruvic Acid
  • Glucose
  • Oxygen