Citrullinaemia type I: a common mutation in the Pacific Island population

J Paediatr Child Health. 2011 May;47(5):262-5. doi: 10.1111/j.1440-1754.2010.01948.x. Epub 2011 Jan 18.

Abstract

Aim: The aim of this study was to develop and apply a mutation screening protocol for the ASS1 gene in order to confirm the diagnosis of citrullinaemia type I in neonates with elevated citrulline on expanded newborn screening (E-NBS).

Methods: Three patients with an elevated citrulline level were identified via routine E-NBS between January and October 2008. Analysis of the ASS1 gene using a polymerase chain reaction and sequencing-based method was successfully applied to all three patients, together with a rapid mutation-specific detection method. Their clinical progress was followed for 16-22 months.

Results: All three patients were homozygous for a previously reported missense mutation, c.787G>A (p.Val263Met), associated with a mild or asymptomatic clinical course.

Conclusions: As a consequence of E-NBS, an increasing number of neonates with elevated citrulline of uncertain clinical significance are being identified. Rapid sequence analysis of the ASS1 gene can be used to confirm citrullinaemia type I and, increasingly, to infer phenotypic severity. Homozygosity for the same mutation was found in all three patients despite non-consanguinity and variable Pacific Island origin. These data suggest that this mutation may be relatively prevalent in these ethnic groups and imply a possible founder effect.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Argininosuccinate Synthase / genetics
  • Argininosuccinate Synthase / isolation & purification
  • Citrulline / blood
  • Citrullinemia / classification*
  • Citrullinemia / diagnosis*
  • Citrullinemia / genetics
  • Citrullinemia / physiopathology
  • Founder Effect
  • Genetic Testing
  • Humans
  • Infant, Newborn
  • Male
  • Mutation / genetics*
  • Pacific Islands

Substances

  • Citrulline
  • Argininosuccinate Synthase