Genome expansion and gene loss in powdery mildew fungi reveal tradeoffs in extreme parasitism

Science. 2010 Dec 10;330(6010):1543-6. doi: 10.1126/science.1194573.

Abstract

Powdery mildews are phytopathogens whose growth and reproduction are entirely dependent on living plant cells. The molecular basis of this life-style, obligate biotrophy, remains unknown. We present the genome analysis of barley powdery mildew, Blumeria graminis f.sp. hordei (Blumeria), as well as a comparison with the analysis of two powdery mildews pathogenic on dicotyledonous plants. These genomes display massive retrotransposon proliferation, genome-size expansion, and gene losses. The missing genes encode enzymes of primary and secondary metabolism, carbohydrate-active enzymes, and transporters, probably reflecting their redundancy in an exclusively biotrophic life-style. Among the 248 candidate effectors of pathogenesis identified in the Blumeria genome, very few (less than 10) define a core set conserved in all three mildews, suggesting that most effectors represent species-specific adaptations.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adaptation, Physiological
  • Ascomycota / genetics*
  • Ascomycota / growth & development
  • Ascomycota / metabolism
  • Ascomycota / pathogenicity
  • Carbohydrate Metabolism
  • Carrier Proteins / genetics
  • Carrier Proteins / metabolism
  • Enzymes / genetics
  • Enzymes / metabolism
  • Evolution, Molecular
  • Fungal Proteins / chemistry
  • Fungal Proteins / genetics
  • Fungal Proteins / metabolism
  • Gene Deletion*
  • Genes, Fungal*
  • Genome, Fungal*
  • Hordeum / microbiology*
  • Host-Pathogen Interactions / genetics
  • Metabolic Networks and Pathways / genetics
  • Molecular Sequence Annotation
  • Plant Diseases / microbiology*
  • Retroelements
  • Sequence Analysis, DNA
  • Species Specificity

Substances

  • Carrier Proteins
  • Enzymes
  • Fungal Proteins
  • Retroelements