Cardiopulmonary effects of matching positive end-expiratory pressure to abdominal pressure in concomitant abdominal hypertension and acute lung injury

J Trauma. 2010 Aug;69(2):375-83. doi: 10.1097/TA.0b013e3181e12b3a.

Abstract

Background: To evaluate the cardiopulmonary effects of positive end-expiratory pressure (PEEP) equalization to intra-abdominal pressure (IAP) in an experimental model of intra-abdominal hypertension (IAH) and acute lung injury (ALI).

Methods: Eight anesthetized pigs were submitted to IAH of 20 mm Hg with a carbon dioxide insufflator for 30 minutes and then submitted to lung lavage with saline and Tween (2.5%). Pressure x volume curves of the respiratory system were performed by a low flow method during IAH and ALI, and PEEP was subsequently adjusted to 27 cm . H2O for 30 minutes.

Results: IAH decreases pulmonary and respiratory system static compliances and increases airway resistance, alveolar-arterial oxygen gradient, and respiratory dead space. The presence of concomitant ALI exacerbates these findings. PEEP identical to AP moderately improved oxygenation and respiratory mechanics; however, an important decline in stroke index and right ventricle ejection fraction was observed.

Conclusions: Simultaneous IAH and ALI produce important impairments in the respiratory physiology. PEEP equalization to AP may improve the respiratory performance, nevertheless with a secondary hemodynamic derangement.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Abdominal Cavity / physiopathology*
  • Acute Lung Injury / physiopathology*
  • Acute Lung Injury / therapy*
  • Animals
  • Compartment Syndromes / physiopathology
  • Compartment Syndromes / therapy
  • Disease Models, Animal
  • Female
  • Heart Function Tests
  • Hemodynamics / physiology
  • Hypertension / physiopathology
  • Positive-Pressure Respiration / methods*
  • Pressure*
  • Probability
  • Random Allocation
  • Respiratory Dead Space*
  • Respiratory Mechanics / physiology
  • Stroke Volume
  • Sus scrofa
  • Vascular Resistance / physiology