Abstract
In myelodysplastic syndromes (MDS), deletions of chromosome 7 or 7q are common and correlate with a poor prognosis. The relevant genes on chromosome 7 are unknown. We report here that EZH2, located at 7q36.1, is frequently targeted in MDS. Analysis of EZH2 deletions, missense and frameshift mutations strongly suggests that EZH2 is a tumor suppressor. As EZH2 functions as a histone methyltransferase, abnormal histone modification may contribute to epigenetic deregulation in MDS.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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DNA-Binding Proteins
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Enhancer of Zeste Homolog 2 Protein
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Genes, Tumor Suppressor
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Histone Methyltransferases
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Histone-Lysine N-Methyltransferase
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Humans
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Mutation*
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Myelodysplastic Syndromes / genetics*
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Polycomb Repressive Complex 2
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Transcription Factors
Substances
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DNA-Binding Proteins
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Transcription Factors
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Histone Methyltransferases
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EZH2 protein, human
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Enhancer of Zeste Homolog 2 Protein
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Histone-Lysine N-Methyltransferase
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Polycomb Repressive Complex 2