Bone marrow stromal cell interaction reduces syndecan-1 expression and induces kinomic changes in myeloma cells

Exp Cell Res. 2010 Jul 1;316(11):1816-28. doi: 10.1016/j.yexcr.2010.03.013. Epub 2010 Mar 20.

Abstract

CD138 (Syndecan 1) is a heparan sulfate proteoglycan that concentrates heparan sulfate-binding growth factors on the surface of normal and malignant plasma cells (multiple myeloma, MMC). Recent studies have shown the presence of a CD138-negative fraction of MMC within myelomatous bone marrow (BM). We employed kinome array technology to characterize this fraction at a molecular level, using a myeloma cell line model. Compared to CD138-positive cells, CD138-negative MMC showed (i) a reduced activity of kinases involved in cell cycle progression, in agreement with a decreased labeling index and (ii) reduced Rho signaling to F-actin. Interestingly, CD138 mRNA and protein expression was reduced upon interaction of MM cells with stromal cell lines and primary mesenchymal cultures, which was accompanied by the acquisition of an increased Bcl6/Blimp1 ratio. Co-culture induced an increased activity of kinases involved in adhesion and a decreased S-phase transition in both CD138-positive and -negative fractions. In addition, CD138-negative MMC demonstrated an increased STAT3 and ERK1/2 activation compared to CD138+ MMC, in agreement with a lower sensitivity to compound exposure. The presence of a less mature, more resistant CD138-negative myeloma cell fraction within bone marrow microniches might contribute to high incidence of relapse of Myeloma patients.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Bone Marrow Cells / pathology*
  • Bone Marrow Cells / physiology*
  • Cell Cycle
  • Cell Differentiation
  • Cell Line
  • Cell Line, Tumor
  • Coculture Techniques
  • Drug Resistance, Neoplasm
  • Humans
  • Multiple Myeloma / drug therapy
  • Multiple Myeloma / genetics
  • Multiple Myeloma / pathology*
  • Multiple Myeloma / physiopathology*
  • Neoplastic Stem Cells / pathology
  • Neoplastic Stem Cells / physiology
  • Phosphotransferases / metabolism
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • RNA, Neoplasm / genetics
  • RNA, Neoplasm / metabolism
  • Recurrence
  • Signal Transduction
  • Stromal Cells / pathology
  • Stromal Cells / physiology
  • Syndecan-1 / genetics
  • Syndecan-1 / metabolism*
  • Transcription Factors / metabolism
  • rho GTP-Binding Proteins / metabolism

Substances

  • RNA, Messenger
  • RNA, Neoplasm
  • SDC1 protein, human
  • Syndecan-1
  • Transcription Factors
  • Phosphotransferases
  • rho GTP-Binding Proteins