Cutting edge: CD28 and c-Rel-dependent pathways initiate regulatory T cell development

J Immunol. 2010 Apr 15;184(8):4074-7. doi: 10.4049/jimmunol.0903933. Epub 2010 Mar 12.

Abstract

Regulatory T cell (Treg) development proceeds via a two-step process in which naive CD4(+) thymocytes are first converted into CD4(+)CD25(+)CD122(+)GITR(+)Foxp3(-) Treg progenitors, followed by a second step in which IL-2 converts these Treg progenitors into CD4(+)Foxp3(+) Tregs. The costimulatory molecule CD28 is required for efficient Treg development. However, the stage at which CD28 affects Treg development remains undefined. In this article, we demonstrate that Cd28(-/-) mice lack Treg progenitors. Furthermore, the P(187)YAP motif in the cytoplasmic tail of CD28, which links CD28 to Lck activation, is required for this process. In contrast, the Y(170)MNM motif, which links CD28 to PI3K activation, is not required for Treg progenitor development. Finally, the CD28/Lck pathway was shown to activate the NF-kappaB family of transcription factors. We demonstrate that c-Rel, but not NF-kappaB1, promotes the development of Treg progenitors. Thus, a CD28/c-Rel-dependent pathway is involved in initiating Treg development.

Publication types

  • Comparative Study
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Motifs / genetics
  • Amino Acid Motifs / immunology
  • Animals
  • CD28 Antigens / biosynthesis
  • CD28 Antigens / genetics
  • CD28 Antigens / physiology*
  • Cell Differentiation / genetics
  • Cell Differentiation / immunology*
  • Cytoplasm / enzymology
  • Cytoplasm / genetics
  • Cytoplasm / immunology
  • Enzyme Activation / genetics
  • Enzyme Activation / immunology
  • Gene Expression Regulation / immunology
  • Gene Knock-In Techniques
  • Interleukin-2 / biosynthesis
  • Lymphocyte Activation / genetics
  • Lymphocyte Activation / immunology
  • Lymphocyte Specific Protein Tyrosine Kinase p56(lck) / genetics
  • Lymphocyte Specific Protein Tyrosine Kinase p56(lck) / metabolism
  • Lymphocyte Specific Protein Tyrosine Kinase p56(lck) / physiology
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Mice, Mutant Strains
  • Mice, Transgenic
  • NF-kappa B / metabolism
  • NF-kappa B p50 Subunit / deficiency
  • NF-kappa B p50 Subunit / genetics
  • NF-kappa B p50 Subunit / physiology
  • Peptide Fragments / genetics
  • Peptide Fragments / physiology
  • Protein Binding / genetics
  • Protein Binding / immunology
  • Proto-Oncogene Proteins c-rel / physiology*
  • Signal Transduction / genetics
  • Signal Transduction / immunology
  • Stem Cells / immunology
  • Stem Cells / metabolism
  • Stem Cells / pathology
  • T-Lymphocytes, Regulatory / cytology*
  • T-Lymphocytes, Regulatory / immunology*
  • T-Lymphocytes, Regulatory / metabolism
  • T-Lymphocytes, Regulatory / pathology

Substances

  • CD28 Antigens
  • Interleukin-2
  • NF-kappa B
  • NF-kappa B p50 Subunit
  • Peptide Fragments
  • Proto-Oncogene Proteins c-rel
  • Nfkb1 protein, mouse
  • Lymphocyte Specific Protein Tyrosine Kinase p56(lck)