Insulin and IGF-I induce pronounced hypertrophy of skeletal myofibers in tissue culture

Am J Physiol. 1991 Mar;260(3 Pt 1):C475-84. doi: 10.1152/ajpcell.1991.260.3.C475.

Abstract

Skeletal myofibers differentiated from primary avian myoblasts in tissue culture can be maintained in positive nitrogen balance in a defined serum-free medium for at least 6-7 days when embedded in a three-dimensional collagen gel matrix. Incubation of established myofiber cultures for 3-7 days with insulin (1 microM) or insulin-like growth factor I (IGF-I, 32 nM) stimulates both cell hyperplasia and myofiber hypertrophy. Mean myofiber diameter increases 71-98%. Insulin-like growth factor II stimulates cell hyperplasia but not myofiber hypertrophy. Cell growth results from a 42-62% increase in total protein synthesis and a 28-38% decrease in protein degradation. Myosin heavy-chain content increases 183-258% because of a 55% stimulation of myosin synthesis and 33-61% inhibition of degradation. Associated with myofiber hypertrophy is a 87-148% increase in the number of myofiber nuclei per unit myofiber length. The results indicate that insulin and IGF-I, but not IGF-II, can induce rapid myofiber hypertrophy in vitro, most likely by stimulating myoblast proliferation and/or fusion to established myofibers.

Publication types

  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Cell Division / drug effects
  • Cells, Cultured
  • Chick Embryo
  • Dose-Response Relationship, Drug
  • Hypertrophy
  • Immunohistochemistry
  • Insulin / pharmacology*
  • Insulin-Like Growth Factor I / pharmacology*
  • Insulin-Like Growth Factor II / pharmacology
  • Kinetics
  • Muscles / cytology*
  • Muscles / drug effects
  • Muscles / ultrastructure
  • Myofibrils / drug effects
  • Myofibrils / ultrastructure
  • Phenylalanine / metabolism
  • Protein Biosynthesis

Substances

  • Insulin
  • Phenylalanine
  • Insulin-Like Growth Factor I
  • Insulin-Like Growth Factor II