Abstract
A flurry of recent revelations is challenging the current dogma on how ubiquitin-dependent processes culminate in the activation of NF-kappaB by TNF. Here, we integrate these findings into a model for TNF-R1 signaling-and underscore the importance of individual components, including linear ubiquitin chains-which allows for the remarkable versatility of the ubiquitin system.
Publication types
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Research Support, Non-U.S. Gov't
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Review
MeSH terms
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Models, Biological*
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NF-kappa B / metabolism
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Receptors, Tumor Necrosis Factor, Type I / metabolism*
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Signal Transduction*
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Tumor Necrosis Factor-alpha / metabolism
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Ubiquitin / metabolism*
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Ubiquitin / physiology
Substances
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NF-kappa B
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Receptors, Tumor Necrosis Factor, Type I
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Tumor Necrosis Factor-alpha
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Ubiquitin