Neurokinin 1 receptor gene polymorphism might be correlated with recurrence rates in endometriosis

Gynecol Endocrinol. 2009 Nov;25(11):726-33. doi: 10.3109/09513590903159631.

Abstract

Introduction: Dysmenorrhoea is the major symptom in women with endometriosis. Recently, pain modulation through Neurokinin-1-receptor (NK1R) pathways have been investigated in neuropathic pain patients. Aim of this study was, therefore, to examine the effect of a single nucleotide polymorphism (SNP) of the NK1R gene on the susceptibility for endometriosis and the disease free survival (DFS) after surgery for endometriosis.

Material and methods: A case-control study was conducted and germline DNA was isolated. Patients were followed up for a recurrence of the disease up to 4 years. Case-control analyses were performed for parameters of the medical history and the genotype of the NK1R-SNP rs881. Furthermore, DFS probabilities were calculated.

Results: Concerning the DFS preoperative pain levels and the NK1R genotype were independent predictors for a recurrence with hazard ratios of 2.55 (95% CI: 1.32-4.95) for patients with a high preoperative pain level and 0.44 for patients with a heterozygous or homozygous variant genotype in rs881 (95% CI: 0.21-0.88).

Conclusion: The polymorphism rs811 seems to be associated with a lower recurrence risk in endometriosis patients. Thus, there might be a clinical relevant role of the NK1 pathway in the pain perception of endometriosis patients.

MeSH terms

  • Adult
  • Case-Control Studies
  • Disease-Free Survival
  • Endometriosis / genetics*
  • Endometriosis / physiopathology
  • Endometriosis / surgery*
  • Female
  • Genetic Predisposition to Disease*
  • Genotype
  • Heterozygote
  • Homozygote
  • Humans
  • Kaplan-Meier Estimate
  • Middle Aged
  • Pain / physiopathology
  • Polymorphism, Single Nucleotide*
  • Preoperative Period
  • Proportional Hazards Models
  • Receptors, Neurokinin-1 / genetics*
  • Secondary Prevention

Substances

  • Receptors, Neurokinin-1