Omega-3 fatty acids, genetic variants in COX-2 and prostate cancer

J Nutrigenet Nutrigenomics. 2009;2(3):149-58. doi: 10.1159/000235565. Epub 2009 Sep 23.

Abstract

Dietary intake of fish and omega-3 polyunsaturated fatty acids (omega-3 PUFAs) may decrease the risk of prostate cancer development and progression to advanced stage disease. This could reflect the anti-inflammatory effects of PUFAs, possibly through mediation of cyclooxygenase (COX), a key enzyme in fatty acid metabolism and inflammation. Despite promising experimental evidence, epidemiological studies have reported somewhat conflicting results regarding the effects of fish/PUFAs on prostate cancer development and progression. The literature suggests that fish, and particularly long-chain omega-3 PUFAs, may have a more pronounced protective effect on biologically aggressive tumors or on their progression, and less on early steps of carcinogenesis. Moreover, the impact of LC omega-3 PUFAs may be modified by variation of the COX-2 gene. Overall, results to date support the hypothesis that long-chain omega-3 PUFAs may impact prostate inflammation and carcinogenesis via the COX-2 enzymatic pathway.

Publication types

  • Research Support, N.I.H., Extramural
  • Review

MeSH terms

  • Animals
  • Cohort Studies
  • Cyclooxygenase 2 / genetics*
  • Cyclooxygenase 2 / metabolism
  • Diet
  • Fatty Acids, Omega-3 / therapeutic use*
  • Fishes
  • Genetic Variation*
  • Humans
  • Inflammation / complications
  • Inflammation / genetics
  • Male
  • Prostatic Neoplasms / enzymology
  • Prostatic Neoplasms / epidemiology
  • Prostatic Neoplasms / genetics*
  • Prostatic Neoplasms / prevention & control
  • Risk Factors

Substances

  • Fatty Acids, Omega-3
  • Cyclooxygenase 2