Abstract
Objective:
To observe the effect of curcumin on protein kinases on cell signal pathway in breast cancer cell MCF-7.
Methods:
The suppressive effect of curcumin on MCF-7 cell proliferation as different concentration and at different time points were observed by MTT assay; The activity of p-ERK, p-NF-kappaB, p-p38 and p-JNK were observed by western blot.
Results:
The suppressive effect of curcumin was shown as a dose dependent and a time dependent manner,and the IC50 was 22.48 micromol/L 40 micromol/L curcumin inhibited the expression of p-ERK, p-NF-kappaB and p-p38, but not p-JNK.
Conclusion:
Curcumin can inhibit MCF-7 cell's proliferation, and its mechanism may be related to the activities of protein kinases on cell signal pathway.
Publication types
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English Abstract
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Research Support, Non-U.S. Gov't
MeSH terms
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Antineoplastic Agents, Phytogenic / pharmacology*
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Blotting, Western
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Breast Neoplasms / metabolism
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Breast Neoplasms / pathology*
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Cell Line, Tumor
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Cell Proliferation / drug effects*
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Cell Survival / drug effects
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Curcuma / chemistry
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Curcumin / pharmacology*
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Dose-Response Relationship, Drug
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Enzyme Inhibitors / pharmacology
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Extracellular Signal-Regulated MAP Kinases / metabolism
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Female
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Humans
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JNK Mitogen-Activated Protein Kinases / metabolism
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NF-kappa B / drug effects
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NF-kappa B / metabolism
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Protein-Tyrosine Kinases / metabolism*
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Signal Transduction / drug effects
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p38 Mitogen-Activated Protein Kinases / metabolism
Substances
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Antineoplastic Agents, Phytogenic
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Enzyme Inhibitors
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NF-kappa B
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Protein-Tyrosine Kinases
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Extracellular Signal-Regulated MAP Kinases
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JNK Mitogen-Activated Protein Kinases
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p38 Mitogen-Activated Protein Kinases
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Curcumin