Persistent body fat mass and inflammatory marker increases after long-term cure of Cushing's syndrome

J Clin Endocrinol Metab. 2009 Sep;94(9):3365-71. doi: 10.1210/jc.2009-0766. Epub 2009 Jun 9.

Abstract

Objective: Although increased central fat mass is characteristic of active Cushing's syndrome (CS), little is known about body composition and secretion of adipokines after long-term recovery of CS. The aim of this study was to evaluate central fat mass and its correlation with adipokines and cardiovascular risk factors in patients after long-term remission of CS.

Methods: Thirty-seven women with CS in remission (27 of pituitary and 10 of adrenal origin; mean age, 50 +/- 14 yr; mean time of hormonal cure, 11 +/- 6 yr) were enrolled and compared to 14 women with active CS and 85 gender-, age-, and body mass index-matched healthy controls. Total and trunk fat mass were measured by dual-energy x-ray absorptiometry scanning. Laboratory parameters and adipokine levels [including adiponectin, visfatin, soluble TNFalpha-receptor 1 (sTNF-R1), sTNF-R2, and IL-6] were measured.

Results: Cured CS patients had more total and trunk fat mass than controls. Cured and active CS had higher levels of sTNF-R1 and IL-6 and lower adiponectin levels than controls. Higher insulin levels and blood pressure in both groups of CS patients and higher apolipoprotein B in cured CS were observed compared to controls. sTNF-R1 correlated positively with percentage of trunk fat mass and remained significant after adjusting for anthropometric parameters.

Conclusion: Despite long-term cure, patients who have suffered CS exhibit persistent accumulation of central fat, as in active hypercortisolemia, with the consequent unfavorable adipokine profile, leading to a state of low-grade inflammation. This situation determines a persistent and increased cardiovascular risk in these patients.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adiponectin / blood
  • Adipose Tissue / metabolism*
  • Adult
  • Aged
  • Blood Pressure
  • Cardiovascular Diseases / etiology
  • Cushing Syndrome / complications
  • Cushing Syndrome / metabolism*
  • Female
  • Humans
  • Inflammation / etiology*
  • Insulin Resistance
  • Interleukin-6 / blood
  • Middle Aged
  • Receptors, Tumor Necrosis Factor, Type I / blood
  • Risk Factors

Substances

  • ADIPOQ protein, human
  • Adiponectin
  • Interleukin-6
  • Receptors, Tumor Necrosis Factor, Type I