The CREB/CREM transcription factors negatively regulate early synaptogenesis and spontaneous network activity

J Neurosci. 2009 Jan 14;29(2):328-33. doi: 10.1523/JNEUROSCI.5252-08.2009.

Abstract

The family of CREB (cAMP response element-binding protein) transcription factors are involved in a variety of biological processes including the development and plasticity of the nervous system. In the maturing and adult brain, CREB genes are required for activity-dependent processes, including synaptogenesis, refinement of connections and long-term potentiation. Here, we use CREB1(Nescre)CREM(-/-) (cAMP-responsive element modulator) mutants to investigate the role of these genes in stimulus-independent patterns of neural activity at early stages. We show that lack of CREB/CREM genes specifically in neural tissue leads to increased synaptogenesis and to a dramatic increase in the levels of spontaneous network activity at embryonic stages. Thus, the functions of CREB/CREM genes in neural activity differ in distinct periods of neural development.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Age Factors
  • Animals
  • Brain / cytology
  • Brain / embryology
  • Brain / metabolism
  • Calcium / metabolism
  • Cyclic AMP Response Element Modulator / deficiency
  • Cyclic AMP Response Element Modulator / physiology*
  • Cyclic AMP Response Element-Binding Protein / deficiency
  • Cyclic AMP Response Element-Binding Protein / physiology*
  • Embryo, Mammalian
  • In Vitro Techniques
  • Mice
  • Mice, Knockout
  • Neural Pathways / physiology*
  • Neural Pathways / ultrastructure
  • Neurons / physiology*
  • Neurons / ultrastructure
  • Synapses / genetics*
  • Synapses / ultrastructure

Substances

  • Creb1 protein, mouse
  • Cyclic AMP Response Element-Binding Protein
  • Cyclic AMP Response Element Modulator
  • Calcium