Significant association between neutrophil aggregation in aspirated thrombus and myocardial damage in patients with ST-segment elevation acute myocardial infarction

Circ J. 2009 Jan;73(1):139-44. doi: 10.1253/circj.cj-08-0609. Epub 2008 Dec 2.

Abstract

Background: This study was designed to clarify the relationship between myocardial damage and platelet-neutrophil aggregation in patients with acute myocardial infarction (AMI).

Methods and results: The study group comprised 107 patients with ST-segment elevation AMI, in whom aspiration catheter was used during emergency percutaneous coronary intervention. Patients were divided into 2 groups according to the cellular density of neutrophils in the aspirated sample: group L (n=53), <or=100 neutrophils/0.025 mm(2) thrombus; group H (n=54), >100 neutrophils/0.025 mm(2) thrombus. Myocardial blush grade (MBG) <or=1 and ST-segment resolution (STR) <50% were more frequently found in group H than in group L. Peak creatine kinase level tended to be higher and left ventricular ejection fraction (LVEF) at 6 months after onset was lower in group H than in group L. Multivariate analysis showed that high neutrophil density in aspirated thrombus was an independent predictor of MBG <or=1, STR <50%, and low LVEF at 6 months after onset.

Conclusions: Platelet-neutrophil aggregates retrieved from ruptured plaque may be associated with impaired coronary microcirculation and resultant myocardial necrosis/dysfunction. These findings underscore the clinical importance of the interaction between thrombosis and inflammation in the pathogenesis of AMI.

MeSH terms

  • Aged
  • Blood Platelets / pathology
  • Cardiac Catheterization
  • Cell Aggregation
  • Coronary Vessels / pathology
  • Coronary Vessels / physiopathology
  • Creatine Kinase / blood
  • Electrocardiography*
  • Female
  • Humans
  • Male
  • Microcirculation / physiology
  • Middle Aged
  • Multivariate Analysis
  • Myocardial Infarction / pathology*
  • Myocarditis / pathology*
  • Neutrophils / pathology*
  • Retrospective Studies
  • Stroke Volume / physiology
  • Thrombosis / pathology*
  • Ventricular Remodeling / physiology

Substances

  • Creatine Kinase