Apolipoprotein E and paraoxonase 1 polymorphisms are associated with lower serum thyroid hormones in postmenopausal women

Clin Endocrinol (Oxf). 2009 Aug;71(2):284-90. doi: 10.1111/j.1365-2265.2008.03476.x. Epub 2008 Nov 9.

Abstract

Objective: Autoimmune thyroiditis and overt or subclinical hypothyroidism have been associated with increased prevalence of cardiovascular disease (CVD).

Design: Cross-sectional investigation of the association between gene polymorphisms related to CVD with thyroid function and autoimmunity.

Patients: In total 84 healthy postmenopausal women aged 49-69 years.

Measurements: FT3, FT4, anti-TPO and anti-TG were assessed in the sera of participants. The following polymorphisms were assessed from peripheral lymphocyte DNA: Apolipoprotein E E2/E3/E4, paraoxonase 1 A/B, Glycoprotein IIIa leu33pro, MTHFR ala222val, ApoBarg3500gln, plasminogen activator inhibitor 1 4G/5G, cholesterol 7-alpha hydroxylase A204C and cholesterol ester transfer protein B1/B2.

Results: A statistically significant correlation was found between Apolipoprotein E and paraoxonase 1 polymorphisms and serum thyroid hormones: carriers of the E2 or E4 allele of the ApoE gene had lower levels of FT4 (P = 0.0005) than women with the E3/E3 genotype. Carriers of the B allele of paraoxonase 1 gene had lower levels of FT3 compared to women with the wild-type genotype (P = 0.047). A statistically significant positive association (P = 0.049) was also observed between anti-TG antibodies and the presence of the E2 allele of the Apolipoprotein E gene.

Conclusions: Polymorphisms of apolipoprotein E and paraoxonase 1 are associated with different levels of thyroid hormone and anti-Tg antibody levels in the study population in this pilot study. The mechanism underlying this association remains to be elucidated.

MeSH terms

  • Aged
  • Apolipoproteins E / genetics*
  • Aryldialkylphosphatase / genetics*
  • Cardiovascular Diseases / epidemiology
  • Cardiovascular Diseases / genetics
  • Cross-Sectional Studies
  • Female
  • Genetic Predisposition to Disease
  • Humans
  • Middle Aged
  • Polymorphism, Genetic*
  • Postmenopause / blood*
  • Postmenopause / genetics
  • Thyroid Hormones / blood*

Substances

  • Apolipoproteins E
  • Thyroid Hormones
  • Aryldialkylphosphatase