Counterbalancing forces: what is thrombospondin-1 doing in atherosclerotic lesions?

Circ Res. 2008 Nov 7;103(10):1053-5. doi: 10.1161/CIRCRESAHA.108.188870.
No abstract available

Publication types

  • Comment
  • Editorial
  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • Apolipoproteins E / genetics
  • Apoptosis / genetics
  • Atherosclerosis / genetics
  • Atherosclerosis / metabolism*
  • Atherosclerosis / pathology
  • Blood Platelets / metabolism
  • Blood Platelets / pathology
  • Elastin / genetics
  • Elastin / metabolism
  • Genotype
  • Macrophages, Peritoneal / metabolism*
  • Macrophages, Peritoneal / pathology
  • Mice
  • Mice, Knockout
  • Myocytes, Smooth Muscle / metabolism*
  • Myocytes, Smooth Muscle / pathology
  • Necrosis / metabolism
  • Necrosis / pathology
  • Phagocytosis / genetics
  • Thrombospondin 1 / genetics

Substances

  • Apolipoproteins E
  • Thrombospondin 1
  • Elastin