Reactive hyperemia and BOLD MRI demonstrate that VEGF inhibition, age, and atherosclerosis adversely affect functional recovery in a murine model of peripheral artery disease

J Magn Reson Imaging. 2008 Oct;28(4):996-1004. doi: 10.1002/jmri.21517.

Abstract

Purpose: To develop magnetic resonace imaging (MRI) methods for functional assessment of arteriogenesis in a murine model of peripheral artery disease to quantify the influences of vascular endothelial growth factor (VEGF), age, and atherosclerosis.

Materials and methods: Reactive hyperemia (RH), which was induced using a device designed for remote and transient occlusion of the aorta and vena cava, was measured by blood-oxygen-level-dependent MRI. Twenty-eight days after femoral artery ligation, peak height (PH) and time to peak (TTP) of the RH response was compared with sham-operated animals in 10-week-old C57Bl6, 9-month-old C57Bl6, and 9-month-old Ldlr(-/-)Apobec(-/-) mice. The contribution of VEGF to functional recovery was assessed in young mice. Angiogenesis was quantified using an anti-PECAM1 radioimmunoassay.

Results: In young animals, angiogenesis was maximal 7 days after ligation, whereas functional recovery took 28 days. Inhibition of VEGF eliminated the angiogenesis seen at 7 days and reduced RH (PH, P < 0.05). At day 28, RH was altered in old (TTP, P < 0.05) and atherosclerotic (PH, P < 0.05; TTP, P < 0.05) animals. RH was different in young, old, and atherosclerotic sham animals. Old and atherosclerotic mice showed reduced angiogenesis.

Conclusion: The method presented herein can provide a sensitive assay for the functional assessment of arteriogenesis and highlights the contribution of VEGF, age, and atherosclerosis to this process.

MeSH terms

  • Age Factors
  • Analysis of Variance
  • Animals
  • Atherosclerosis / metabolism
  • Atherosclerosis / physiopathology*
  • Disease Models, Animal
  • Hindlimb
  • Hyperemia / metabolism
  • Hyperemia / physiopathology*
  • Magnetic Resonance Imaging / methods*
  • Mice
  • Neovascularization, Physiologic / drug effects*
  • Peripheral Vascular Diseases / metabolism
  • Peripheral Vascular Diseases / physiopathology*
  • Radioimmunoassay
  • Recovery of Function
  • Risk Factors
  • Time Factors
  • Vascular Endothelial Growth Factor A / antagonists & inhibitors*

Substances

  • Vascular Endothelial Growth Factor A