Lateralization of 17beta-hydroxysteroid dehydrogenase type 10 in hippocampi of demented and psychotic people

Dement Geriatr Cogn Disord. 2008;26(3):193-8. doi: 10.1159/000151778. Epub 2008 Sep 3.

Abstract

Objective: The multifunctional mitochondrial enzyme 17beta-hydroxysteroid dehydrogenase type 10 could play a role in the development of Alzheimer disease via its high-affinity binding to amyloid-beta peptides and its overexpression.

Methods: We evaluated the specificity of alterations in mRNA/enzyme expression levels in human right and left hippocampi.

Results: We observed a trend towards right/left laterality in nondemented nonpsychotic controls; however, the degree of asymmetry was higher for mRNA when compared to enzyme expression levels. In Alzheimer disease and schizophrenia, significant shifts to left/right asymmetry were found and the changes were associated with more marked increases in mRNA/enzyme expression in the left hemisphere. On the other hand, no alterations were observed in people with multi-infarct dementia.

Conclusion: Our results support studies reporting an impairment of mitochondria in Alzheimer disease or schizophrenia and a higher vulnerability of the dominant hemisphere to pathological processes. Overexpression of the enzyme could be used to distinguish Alzheimer disease from multi-infarct dementia.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 3-Hydroxyacyl CoA Dehydrogenases / genetics*
  • 3-Hydroxyacyl CoA Dehydrogenases / metabolism
  • Aged
  • Aged, 80 and over
  • Alzheimer Disease / pathology
  • Alzheimer Disease / physiopathology*
  • Amyloid beta-Peptides / metabolism
  • Cerebral Infarction / pathology
  • Cerebral Infarction / physiopathology
  • Female
  • Functional Laterality / physiology*
  • Gene Expression Regulation, Enzymologic
  • Hippocampus / enzymology*
  • Hippocampus / pathology
  • Humans
  • Male
  • Middle Aged
  • Mitochondria / enzymology
  • Psychotic Disorders / pathology
  • Psychotic Disorders / physiopathology*
  • RNA, Messenger / metabolism

Substances

  • Amyloid beta-Peptides
  • RNA, Messenger
  • 3-Hydroxyacyl CoA Dehydrogenases
  • HSD17B10 protein, human