Functional recovery after spinal cord injury in mice through activation of microglia and dendritic cells after IL-12 administration

J Neurosci Res. 2008 Jul;86(9):1972-80. doi: 10.1002/jnr.21658.

Abstract

We have previously reported that the transplantation of dendritic cells (DCs) brings about functional recovery after spinal cord injury in mice through the activation of endogenous microglia/macrophages and neural stem/progenitor cells. In this study, the effect of interleukin-12 (IL-12), which is secreted from DCs, was evaluated for the treatment of spinal cord injury in mice. Administration of IL-12 into the injured site significantly increased the number of activated microglia/macrophages and DCs as well as the expression of brain-derived neurotrophic factor surrounding the lesion site. Immunohistochemical analyses showed that de novo neurogenesis and remyelination were induced by IL-12 treatment. Furthermore, an open field test using Basso-Beattie-Brenham scoring revealed a significant improvement of locomotor function in mice treated with IL-12. These results suggest that IL-12 administration into the injured spinal cord results in a functional recovery through the activation of microglia/macrophages and DCs.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Bromodeoxyuridine
  • Dendritic Cells / physiology*
  • Female
  • Hindlimb
  • Immunohistochemistry
  • Interleukin-12 / pharmacology
  • Mice
  • Mice, Inbred BALB C
  • Mice, Inbred C57BL
  • Microglia / physiology*
  • Motor Activity / drug effects
  • Motor Activity / physiology
  • Myelin Sheath / drug effects
  • Myelin Sheath / physiology
  • Spinal Cord / drug effects
  • Spinal Cord / physiopathology
  • Spinal Cord Injuries / rehabilitation*

Substances

  • Interleukin-12
  • Bromodeoxyuridine