Comparison of expression profiles induced by dust mite in airway epithelia reveals a common pathway

Allergy. 2008 Apr;63(4):461-7. doi: 10.1111/j.1398-9995.2007.01621.x.

Abstract

Background: Airway epithelial cells have shown to be active participants in the defense against pathogens by producing signaling and other regulatory molecules in response to the encounter.

Methods: In previous manuscripts, we have studied the effect of house dust mite (HDM) extract on both an epithelial cell-line (H292) and primary nasal epithelial cell. When we compare these responses we conclude that the H292 cells more closely resemble nasal epithelium of healthy controls (share 107 probe-sets) than of allergic individuals (share 17 probe-sets).

Results: Interestingly, probably because of an absent intraindividual variation between samples, more probe-sets (8280) change expression significantly in H292 than in either healthy (555) or allergic (401) epithelium.

Conclusions: A direct comparison of all the responses in these epithelial cells reveals a core-response to HDM of just 29 genes. These genes (CCL20, IL-8, CXCL2, CXCL1, IL-1B, AREG, TNFAIP3, HBEGF, PTGS2, BMP2, LDLR, PLAUR, PLAU, NFKB2, NFKB1, JUN, ATF3, EGR1, NPC1, TICAM1, EPHA2, CTGF, DUSP1, SPRY1, TLR-3, complement factor C3, IVNS1ABP, SerpinB3, and PSAT1) have described links with allergy or inflammation and may even describe the well-established relationship between viral infections and allergic exacerbations or allergy development.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antigens, Dermatophagoides / immunology*
  • Cell Line, Tumor
  • Cells, Cultured
  • Epithelial Cells / immunology*
  • Gene Expression Profiling
  • Humans
  • Hypersensitivity / genetics*
  • Hypersensitivity / immunology
  • Nasal Mucosa / cytology
  • Nasal Mucosa / immunology*
  • Oligonucleotide Array Sequence Analysis
  • Pyroglyphidae / immunology

Substances

  • Antigens, Dermatophagoides