Abstract
Five highly amikacin-resistant Acinetobacter baumannii isolates were collected at a medical center in Pennsylvania. The aminoglycoside resistance was due to the production of the 16S rRNA methylase ArmA. Two of the isolates coproduced OXA-23 beta-lactamase and were highly resistant to carbapenems as well. The isolates were genetically closely related by pulsed-field gel electrophoresis.
Publication types
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Research Support, N.I.H., Extramural
MeSH terms
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Acinetobacter Infections / microbiology
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Acinetobacter baumannii / drug effects
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Acinetobacter baumannii / enzymology*
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Acinetobacter baumannii / genetics
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Amikacin / pharmacology
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Aminoglycosides / pharmacology
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Anti-Bacterial Agents / pharmacology
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Carbapenems / pharmacology
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Electrophoresis, Gel, Pulsed-Field
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Humans
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Methyltransferases / genetics
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Methyltransferases / metabolism*
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Microbial Sensitivity Tests
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Molecular Sequence Data
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North America
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RNA, Ribosomal, 16S / metabolism*
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Sequence Analysis, DNA
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beta-Lactam Resistance / genetics
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beta-Lactamases / metabolism
Substances
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Aminoglycosides
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Anti-Bacterial Agents
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Carbapenems
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RNA, Ribosomal, 16S
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Amikacin
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Methyltransferases
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beta-lactamase OXA-23
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beta-Lactamases