IL-7-mediated protection against minor-antigen-mismatched allograft rejection is associated with enhanced recovery of regulatory T cells

Haematologica. 2007 Aug;92(8):1099-106. doi: 10.3324/haematol.10625.

Abstract

Background and objectives: Interleukin-7 (IL-7) has been studied for its possible immunorestorative capacities following stem cell transplantation and has been shown to enhance post-transplant immune recovery predominantly by peripheral T-cell expansion. A major concern of IL-7 is its possible aggravating effect on graft-versus-host and host-versus-graft reactivity.

Design and methods: To study the effect of IL-7 on host-versus-graft reactivity, we applied IL-7 in an experimental transplantation model using RAG-1-/- mice supplied with B6 CD45.1 congenic T cells as recipients of T-cell depleted allogeneic bone marrow grafts.

Results: Rejection of minor antigen-mismatched bone marrow was significantly reduced in IL-7 treated recipients compared with PBS treated control mice. Rejection was observed in 2 out of 18 IL-7 treated mice compared with 9 out of 17 PBS treated mice (11% vs. 53%; p=0.012). IL-7 administration resulted in enhanced recovery of peripheral blood CD4+CD25+ regulatory T cells (Treg) with a concomitant increase in peripheral blood Foxp3 mRNA expression. IL-7Ra (CD127) was expressed by the vast majority of CD4+Foxp3+ T cells. The incidence of graft rejection following fully MHC mismatched bone marrow transplantation was not reduced nor enhanced by IL-7 administration.

Interpretation and conclusions: Post-transplant IL-7 administration protects against minor antigen-mismatched bone marrow rejection, which may be due to enhanced Treg recovery.

MeSH terms

  • Animals
  • Animals, Congenic
  • Bone Marrow Transplantation / immunology*
  • CD4 Antigens / analysis
  • Drug Evaluation, Preclinical
  • Forkhead Transcription Factors / biosynthesis
  • Forkhead Transcription Factors / genetics
  • Graft Rejection / immunology
  • Graft Rejection / prevention & control*
  • Host vs Graft Reaction / drug effects*
  • Interleukin-2 Receptor alpha Subunit / analysis
  • Interleukin-7 / pharmacology
  • Interleukin-7 / therapeutic use*
  • Interleukin-7 Receptor alpha Subunit / biosynthesis
  • Interleukin-7 Receptor alpha Subunit / genetics
  • Lymphocyte Count
  • Lymphocyte Depletion
  • Mice
  • Mice, Inbred Strains
  • Mice, Knockout
  • Minor Histocompatibility Antigens / immunology*
  • Recombinant Proteins / therapeutic use
  • Specific Pathogen-Free Organisms
  • T-Lymphocytes, Regulatory / drug effects
  • T-Lymphocytes, Regulatory / immunology*
  • Transplantation, Homologous

Substances

  • CD4 Antigens
  • Forkhead Transcription Factors
  • Foxp3 protein, mouse
  • Interleukin-2 Receptor alpha Subunit
  • Interleukin-7
  • Interleukin-7 Receptor alpha Subunit
  • Minor Histocompatibility Antigens
  • Recombinant Proteins