Sensitive ChIP-DSL technology reveals an extensive estrogen receptor alpha-binding program on human gene promoters

Proc Natl Acad Sci U S A. 2007 Mar 20;104(12):4852-7. doi: 10.1073/pnas.0700715104. Epub 2007 Mar 14.

Abstract

ChIP coupled with microarray provides a powerful tool to determine in vivo binding profiling of transcription factors to deduce regulatory circuitries in mammalian cells. Aiming at improving the specificity and sensitivity of such analysis, we developed a new technology called ChIP-DSL using the DNA selection and ligation (DSL) strategy, permitting robust analysis with much reduced materials compared with standard procedures. We profiled general and sequence-specific DNA binding transcription factors using a full human genome promoter array based on the ChIP-DSL technology, revealing an unprecedented number of the estrogen receptor (ERalpha) target genes in MCF-7 cells. Coupled with gene expression profiling, we found that only a fraction of these direct ERalpha target genes were highly responsive to estrogen and that the expression of those ERalpha-bound, estrogen-inducible genes was associated with breast cancer progression in humans. This study demonstrates the power of the ChIP-DSL technology in revealing regulatory gene expression programs that have been previously invisible in the human genome.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Breast Neoplasms / genetics
  • Cell Line, Tumor
  • Chromatin Immunoprecipitation / methods*
  • Estradiol / pharmacology
  • Estrogen Receptor alpha / metabolism*
  • Female
  • Gene Expression Regulation, Neoplastic / drug effects
  • Genome, Human / drug effects
  • Genome, Human / genetics
  • Histones / metabolism
  • Humans
  • Oligonucleotide Array Sequence Analysis / methods*
  • Promoter Regions, Genetic / drug effects
  • Promoter Regions, Genetic / genetics*
  • Protein Binding / drug effects

Substances

  • Estrogen Receptor alpha
  • Histones
  • Estradiol