[VZV-related myelitis: a pathophysiological hypothesis]

Rev Neurol (Paris). 2007 Jan;163(1):89-92. doi: 10.1016/s0035-3787(07)90359-8.
[Article in French]

Abstract

Introduction: Complications of VZV infection in the central nervous system are multiple. VZV-related myelitis is an uncommon complication of herpes zoster.

Observation: We report the case of a 55-year old man with intercostal herpes zoster who presented a subacute medullar syndrome. MRI demonstrated an extended cervico-thoracic medullar hyperintensity on the T2-weighted images. Cerebrospinal fluid (CSF) analysis showed 100 leukocytes/mm3, 0.94 g/L protein, negative VZV PCR, elevated rate of anti-VZV IgG and no oligoclonal bands. Clinical, biological and radiological presentations were compatible with the diagnosis of VZV-related myelitis with three potential pathophysiological mechanisms: infectious, immune post-infectious, vascular. The course was partially favorable after a 3-day regimen of corticosteroid and 3 weeks of acyclovir infusions.

Discussion: Parainfectious myelitis is often the consequence of a viral infection with a post-infectious pathogenesis. Most often, the clinical outcome is good. In this case report, we highlight the VZV vascular tropism and its more severe outcome.

Conclusion: VZV-related myelitis should be diagnosed early. The combination of aciclovir and corticoids infusions seems to be beneficial.

Publication types

  • Case Reports
  • English Abstract

MeSH terms

  • Herpes Zoster*
  • Humans
  • Male
  • Middle Aged
  • Myelitis / physiopathology*
  • Myelitis / virology*