Genetic abnormalities and CNS tumors: report of two cases of ependymoma associated with Klinefelter's Syndrome (KS)

Childs Nerv Syst. 2007 Feb;23(2):219-23. doi: 10.1007/s00381-006-0179-3. Epub 2006 Oct 13.

Abstract

Objects: Genetic syndromes associated with ependymoma are uncommon, with the exception of NF2. We describe two cases of ependymoma presenting with Klinefelter's Syndrome (KS) as co-morbid condition.

Materials and methods: The first patient was diagnosed for KS during pregnancy; he also presented a thyroid agenesis and a deficit of methyltetrahydrofolate reductase (MTHFR); at 30 months of age he was operated on for a grade II ependymoma of IV ventricle; after a multiple-stage surgery, he underwent oral chemotherapy and stereotactic radiotherapy, but after 15 months he presented a local recurrence and died. The second patient was diagnosed for KS at the age of 16 months; at 10 years of age, due to back pain, he underwent an MRI, which showed a cauda equine tumor. He underwent surgery and radiotherapy. Histology was of mixopapillary ependymoma.

Conclusion: In a review of literature, various neoplasms have been described in association with KS. To our knowledge, these are the first two cases reported of ependymoma associated to KS. A retrospective study of 44 monoinstitutional ependymoma cases demonstrated association with genetic syndromes in 22%.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Central Nervous System Neoplasms / complications
  • Central Nervous System Neoplasms / genetics*
  • Child
  • Child, Preschool
  • Ependymoma / complications
  • Ependymoma / genetics*
  • Glial Fibrillary Acidic Protein / metabolism
  • Humans
  • Klinefelter Syndrome / complications
  • Klinefelter Syndrome / genetics*
  • Male
  • Methylenetetrahydrofolate Reductase (NADPH2) / deficiency*

Substances

  • Glial Fibrillary Acidic Protein
  • Methylenetetrahydrofolate Reductase (NADPH2)