TTF-1, a homeodomain-containing transcription factor, regulates feeding behavior in the rat hypothalamus

Biochem Biophys Res Commun. 2006 Oct 27;349(3):969-75. doi: 10.1016/j.bbrc.2006.08.147. Epub 2006 Sep 1.

Abstract

TTF-1 is a member of the NKx family of homeodomain genes, and is required for morphogenesis and fetal diencephalon development. Our previous studies have shown that TTF-1 expression is maintained in some regions of the postnatal rat brain and transactivates the gene expression of several neuropeptides. In this study, a potential role for TTF-1 in the regulation of feeding behavior was identified. Immunohistochemical analysis showed that TTF-1 is present in several hypothalamic nuclei of the adult rat brain involved in the control of feeding behavior. Food deprivation for two days markedly increased the hypothalamic levels of TTF-1 mRNA and protein. Intracerebroventricular administration of an antisense TTF-1 oligodeoxynucleotide significantly decreased TTF-1 protein abundance in the hypothalamus. This TTF-1 decrease was followed by a significant decrease in neuropeptide Y mRNA content and an increase in proopiomelanocortin mRNA content, and in turn resulted in a decrease of the animal's food intake and body weight. These results suggest a novel role for TTF-1 in the regulation of feeding behavior in the rat hypothalamus.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Body Weight / drug effects
  • Feeding Behavior* / drug effects
  • Hypothalamus / drug effects
  • Hypothalamus / metabolism*
  • Injections, Intravenous
  • Male
  • Neuropeptide Y / genetics
  • Nuclear Proteins / genetics
  • Nuclear Proteins / metabolism*
  • Oligodeoxyribonucleotides, Antisense / pharmacology
  • Pro-Opiomelanocortin / genetics
  • RNA, Messenger / genetics
  • Rats
  • Rats, Sprague-Dawley
  • Thyroid Nuclear Factor 1
  • Transcription Factors / genetics
  • Transcription Factors / metabolism*

Substances

  • Neuropeptide Y
  • Nkx2-1 protein, rat
  • Nuclear Proteins
  • Oligodeoxyribonucleotides, Antisense
  • RNA, Messenger
  • Thyroid Nuclear Factor 1
  • Transcription Factors
  • Pro-Opiomelanocortin