Dietary fish oil reduces the occurrence of early afterdepolarizations in pig ventricular myocytes

J Mol Cell Cardiol. 2006 Nov;41(5):914-7. doi: 10.1016/j.yjmcc.2006.08.001. Epub 2006 Sep 11.

Abstract

Fish oil reduces sudden cardiac death in post myocardial infarction patients. Life-threatening arrhythmias in heart failure are associated with repolarization abnormalities leading to EAD(1) formation. We examined the effects of incorporated fish oil omega3-PUFAs(2) on EAD formation in pig myocytes. Pigs were fed a diet rich in fish oil or sunflower oil (control) for 8 weeks. Myocytes were isolated by enzymatic dissociation and patch-clamped. Susceptibility to EAD formation was tested using E4031 (5 microM), a blocker of I(Kr). The fish oil diet in pigs resulted in increased incorporation of omega3-PUFAs in the sarcolemma of the myocytes compared to the control diet and caused a reduced occurrence of E4031-induced EADs in pig myocytes. A shorter action potential, a reduced action potential prolongation in response to E-4031 and a reduced reactivation of I(Ca,L) by omega3-PUFAs may explain the observed reduction in EADs. A diet rich in fish oil protects against EAD formation.

Publication types

  • Evaluation Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Action Potentials*
  • Animals
  • Fatty Acids, Omega-3 / pharmacology*
  • Fish Oils / pharmacology*
  • Heart Ventricles / cytology
  • Heart Ventricles / drug effects*
  • Male
  • Myocytes, Cardiac / drug effects*
  • Swine
  • Ventricular Function

Substances

  • Fatty Acids, Omega-3
  • Fish Oils