Breast cancer cells secreted platelet-derived growth factor-induced motility of vascular smooth muscle cells is mediated through neuropilin-1

Mol Carcinog. 2006 Nov;45(11):871-80. doi: 10.1002/mc.20248.

Abstract

Motility of vascular smooth muscle cells (SMCs) is an essential step for both normal and pathologic angiogenesis. We report here that breast tumor cells, such as MCF-7 and MDA-MB-231, can modulate this SMC migration. We present evidence that the tumor cell-derived platelet-derived growth factor (PDGF) is the key regulator of vascular SMCs motility induced by breast cancer cells. PDGF significantly upregulates neuropilin-1 (NRP-1) mRNA expression and protein production in aortic smooth muscle cells (AOSMCs) and depletion of NRP-1 production by AOSMCs with specific short hairpin RNA (shRNA) prevents the PDGF-dependent migration of vascular SMCs. Moreover, we demonstrate that PDGF physically interacts with NRP-1. We propose that tumor-derived PDGF and NRP-1 of AOSMCs function as a relay system that promotes motility of vascular SMCs.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Base Sequence
  • Breast Neoplasms / metabolism*
  • Breast Neoplasms / pathology
  • Cell Line, Tumor
  • Cell Movement / physiology*
  • Cells, Cultured
  • Culture Media, Conditioned
  • DNA Primers
  • Humans
  • Muscle, Smooth, Vascular / cytology*
  • Neuropilin-1 / physiology*
  • Platelet-Derived Growth Factor / physiology*
  • Reverse Transcriptase Polymerase Chain Reaction

Substances

  • Culture Media, Conditioned
  • DNA Primers
  • Platelet-Derived Growth Factor
  • Neuropilin-1