Further evidence of association of OPRD1 & HTR1D polymorphisms with susceptibility to anorexia nervosa

Biol Psychiatry. 2007 Feb 1;61(3):367-73. doi: 10.1016/j.biopsych.2006.04.007. Epub 2006 Jun 27.

Abstract

Background: A recent study reported strong evidence for the involvement of a region on human chromosome 1 and genetic susceptibility to anorexia nervosa (AN). A more detailed analysis of this region has suggested 2 genes that may account for this susceptibility. These data suggest that polymorphisms in both the serotonin 1D (HTR1D) and opioid delta 1 (OPRD1) receptor genes show a significant association with restricting AN (RAN).

Methods: In the current study, we have conducted an independent association study on 226 females meeting DSM-IV criteria for AN and 678 matched volunteers.

Results: We genotyped 4 SNPs in HTR1D and 6 SNPs in OPRD1. 3 SNPs were found to be associated with both RAN and binge-purge AN (BPAN) within the gene for OPRD1. We also found evidence of association between 2 polymorphisms within HTR1D and RAN.

Conclusions: These data support the hypothesis that polymorphisms within this region form a component of the genetic basis to susceptibility to RAN. However, further work is required to understand the processes that may be mediated by these genes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Alleles
  • Anorexia Nervosa / epidemiology
  • Anorexia Nervosa / genetics*
  • Anorexia Nervosa / psychology
  • DNA / genetics
  • Female
  • Gene Frequency
  • Genotype
  • Humans
  • Linkage Disequilibrium / genetics
  • Odds Ratio
  • Polymorphism, Genetic / genetics*
  • Psychiatric Status Rating Scales
  • Receptor, Serotonin, 5-HT1D / genetics*
  • Receptors, Opioid, delta / genetics*
  • United Kingdom / epidemiology

Substances

  • Receptor, Serotonin, 5-HT1D
  • Receptors, Opioid, delta
  • DNA