CD45 negatively regulates tumour necrosis factor and interleukin-6 production in dendritic cells

Immunology. 2006 Jun;118(2):250-6. doi: 10.1111/j.1365-2567.2006.02363.x.

Abstract

CD45 is known to regulate signalling through many different surface receptors in diverse haemopoietic cell types. Here we report for the first time that CD45-/- bone marrow dendritic cells (BMDC) are more activated than CD45+/+ cells and that tumour necrosis factor (TNF) and interleukin-6 (IL-6) production by BMDC and splenic dendritic cells (sDC), is increased following stimulation via Toll-like receptor (TLR)3 and TLR9. Nuclear factor-kappaB activation, an important downstream consequence of TLR3 and TLR9 signalling, is also increased in CD45-/- BMDC. BMDC of CD45-/- mice also produce more TNF and IL-6 following stimulation with the cytokines TNF and interferon-alpha. These results show that TLR signalling is increased in CD45-/- dendritic cells and imply that CD45 is a negative regulator of TLR and cytokine receptor signalling in dendritic cells.

MeSH terms

  • Animals
  • Bone Marrow Cells / immunology
  • Cells, Cultured
  • CpG Islands / immunology
  • Cytokines / immunology
  • Dendritic Cells / immunology*
  • Interferon Inducers / immunology
  • Interleukin-6 / biosynthesis*
  • Leukocyte Common Antigens / immunology*
  • Mice
  • Mice, Inbred C57BL
  • NF-kappa B / metabolism
  • Poly I-C / immunology
  • Signal Transduction
  • Spleen / immunology
  • Toll-Like Receptor 3 / immunology
  • Toll-Like Receptor 9 / immunology
  • Tumor Necrosis Factor-alpha / biosynthesis*

Substances

  • Cytokines
  • Interferon Inducers
  • Interleukin-6
  • NF-kappa B
  • Tlr9 protein, mouse
  • Toll-Like Receptor 3
  • Toll-Like Receptor 9
  • Tumor Necrosis Factor-alpha
  • Leukocyte Common Antigens
  • Poly I-C