Effects of D-galactose on the expression of hippocampal peripheral-type benzodiazepine receptor and spatial memory performances in rats

Psychoneuroendocrinology. 2006 Aug;31(7):805-11. doi: 10.1016/j.psyneuen.2006.03.004. Epub 2006 May 16.

Abstract

The changes in spatial memory performances and the binding of hippocampal peripheral-type benzodiazepine receptor (PBR) induced by D-galactose (D-gal) were investigated in rats. The animals were randomly divided into two groups: saline-treated group and D-gal-induced aging group. All rats received 56 days of injection followed by 5 days of behavioral tests. The D-gal-induced aging rats presented significant impairment in water maze performance, compared with that in the saline-treated rats. A significant decrease in [3H]PK11195 binding in the synaptosomes from hippocampus in the D-gal-induced aging rats was observed, compared to that in the saline-treated rats. Meanwhile, the Scatchard analysis revealed that there was a decrease in Bmax, with no significant change in KD. Further analysis demonstrated that water maze performance was closely related to the PK11195 binding in all rats. These results suggest that D-gal decreased the density of PBR in hippocampal synaptosomes, which may be attributable to the progressive pathogenesis of aging in rats.

Publication types

  • Comparative Study
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Aging / drug effects
  • Aging / metabolism
  • Animals
  • Female
  • Galactose / metabolism
  • Galactose / pharmacology*
  • Hippocampus / drug effects
  • Hippocampus / metabolism*
  • Isoquinolines / metabolism
  • Male
  • Maze Learning / drug effects
  • Maze Learning / physiology*
  • Models, Animal
  • Neurotoxins / pharmacology
  • Random Allocation
  • Rats
  • Rats, Sprague-Dawley
  • Receptors, GABA-A / classification
  • Receptors, GABA-A / drug effects
  • Receptors, GABA-A / metabolism*
  • Spatial Behavior / drug effects
  • Spatial Behavior / physiology
  • Synaptosomes / drug effects
  • Synaptosomes / metabolism*

Substances

  • Isoquinolines
  • Neurotoxins
  • Receptors, GABA-A
  • Galactose
  • PK 11195