Regulation of genes involved in dopamine transporter modulation by acute cocaine in rat striatum

Neurosci Lett. 2006 May 8;398(3):235-40. doi: 10.1016/j.neulet.2006.01.001. Epub 2006 Feb 3.

Abstract

It is well established that acute administration of psychostimulants alters dopamine transport. However, the exact mechanism of this modulation is still unknown. In this study we examined the mRNA levels of several proteins involved in the various proposed processes following cocaine administration. The expression levels of several immediate early genes were also studied. This was performed in rat striatum using real-time quantitative PCR. As expected, a marked increase of the immediate early genes Fos, Egr1 and Egr3 was observed. Egr2 was also found up-regulated. Among the different genes studied only Synaptotagmin4 in the SNARE family and Synphilin1 in the synaptic vesicles binding family were modulated by acute cocaine treatment. Interestingly, acute amphetamine treatment did not increase either Synaptotagmin4 and Synphilin1 mRNA levels, although increases in early genes expression were noted.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amphetamine / pharmacology
  • Animals
  • Central Nervous System Stimulants / pharmacology*
  • Clathrin / physiology
  • Cocaine / pharmacology*
  • Corpus Striatum / drug effects*
  • Corpus Striatum / metabolism
  • Dopamine Plasma Membrane Transport Proteins / genetics
  • Dopamine Plasma Membrane Transport Proteins / metabolism*
  • Endocytosis
  • Genes, Immediate-Early*
  • Male
  • Protein Binding
  • RNA, Messenger / biosynthesis
  • Rats
  • Rats, Sprague-Dawley
  • Synaptic Vesicles / metabolism

Substances

  • Central Nervous System Stimulants
  • Clathrin
  • Dopamine Plasma Membrane Transport Proteins
  • RNA, Messenger
  • Amphetamine
  • Cocaine