Lrrk2 and Lewy body disease

Ann Neurol. 2006 Feb;59(2):388-93. doi: 10.1002/ana.20731.

Abstract

Objective: The Lrrk2 kinase domain G2019S substitution is the most common genetic basis of familial and sporadic parkinsonism. Patients harboring the G2019S substitution usually present with clinical Parkinson's disease.

Methods: Herein, we report that the most common neuropathology of G2019S-associated Parkinson's disease is Lewy body disease.

Results: Lrrk2 G2019S was observed in approximately 2% (n = 8) of our Parkinson's disease/Lewy body disease cases (n = 405). The mutation was also found in one control subject and one Alzheimer's disease patient, reflecting reduced penetrance.

Interpretation: Therapeutic strategies targeted at modulating Lrrk2 kinase activity may be important to treat patients with genetically defined familial or typical sporadic Parkinson's disease.

Publication types

  • Comparative Study
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Aged, 80 and over
  • Brain Stem / metabolism
  • Brain Stem / pathology
  • DNA Mutational Analysis / methods
  • Female
  • Glycine / genetics
  • Humans
  • Leucine-Rich Repeat Serine-Threonine Protein Kinase-2
  • Lewy Body Disease / genetics*
  • Male
  • Models, Molecular
  • Mutation*
  • Protein Serine-Threonine Kinases / genetics*
  • Proteins / genetics

Substances

  • Proteins
  • serin-rich spermatogenic protein 1, rat
  • LRRK2 protein, human
  • Leucine-Rich Repeat Serine-Threonine Protein Kinase-2
  • Protein Serine-Threonine Kinases
  • Glycine